Consider reports of pain, noting location and intensity (scale of 0–10). Note precipitating factors and nonverbal pain cues. | Favorable in determining pain management needs and effectiveness of program. |
Recommend or provide firm mattress or bedboard, small pillow. Elevate linens with bed cradle as needed. | Soft and sagging mattress, large pillows prevent maintenance of proper body alignment, placing stress on affected joints. Elevation of bed linens reduces pressure on inflamed or painful joints. |
Suggest patient assume position of comfort while in bed or sitting in chair. Promote bedrest as indicated. | In severe disease or acute exacerbation, total bedrest may be necessary (until objective and subjective improvements are noted) to limit pain or injury to joint. |
Place and monitor use of pillows, sandbags, trochanter rolls, splints, braces. | Rests painful joints and maintains neutral position. Note: Use of splints can decrease pain and may reduce damage to joint; however, prolonged inactivity can result in loss of joint mobility and function. |
Encourage frequent changes of position. Assist patient to move in bed, supporting affected joints above and below, avoiding jerky movements. | Prevents general fatigue and joint stiffness. Stabilizes joint, decreasing joint movement and associated pain. |
Monitor the duration, not the intensity, of morning stiffness. | Duration more accurately reflects the disease’s severity. |
Recommend that patient take warm bath or shower upon arising or at bedtime. Apply warm, moist compresses to affected joints several times a day. Monitor water temperature of compress, baths, and so on. | Heat promotes muscle relaxation and mobility, decreases pain, and relieves morning stiffness. Sensitivity to heat may be diminished and dermal injury may occur. |
Provide gentle massage. | Promotes relaxation and reduces muscle tension. |
Encourage use of stress management techniques such as progressive relaxation, biofeedback, visualization, guided imagery, self-hypnosis, and controlled breathing. Provide Therapeutic Touch. | Promotes relaxation, provides sense of control, and may enhance coping abilities. |
Involve in diversional activities appropriate for individual situation. | Refocuses attention, provides stimulation, and enhances self-esteem and feelings of general well-being. |
Medicate before planned activities and exercises as indicated. | Promotes relaxation, reduces muscle tension and spasms, facilitating participation in therapy. |
Administer medications as indicated: |
- Salicylates: aspirin (ASA) (Acuprin, Ecotrin, ZORprin)
| ASA exerts an anti-inflammatory and mild analgesic effect, decreasing stiffness and increasing mobility. ASA must be taken regularly to sustain a therapeutic blood level. Research indicates that ASA has the lowest toxicity index of commonly prescribed NSAIDs. |
- Nonsalicylates (NSAIDs):ibuprofen (Advil, Motrin), naproxen (Aleve, Naprosyn), sulindac (Clinoril), prioxicam (Feldene), fenoprofen (Nalfon), diclofenac (Voltaren), ketoprofen (Orudis), ketorolac (Toradol), nabumetone (Relafen)
| These drugs control mild to moderate pain and inflammation by inhibition of prostaglandin synthesis. |
- Glucocorticoids:prednisone (Deltasone), methylprednisolone (Depo-Medrol), dexamethasone (Decadron)
| These drugs modify the immune response and suppress inflammation. |
- Disease-modifying antirheumatic drugs (DMARD): methotrexate (Rheumatrex), hydroxychloroquine (Plaquenil), sulfasalazine (Azulfidine), gold compounds, auranofin (Ridaura), azathioprine (Imuran), leflunomide (Arava)
| These drugs vary in action, but all reduce pain and swelling, lessening arthritic symptoms rather than eliminating them. Arava (FDA approved in 1998) is the first oral drug shown to slow progression of RA and damage to joints. |
- COX-2 inhibitors:celecoxib (Celebrex), rofecoxib (Vioxx)
| A new class of medication, COX-2 inhibitors interfere with prostaglandin production, similarly to NSAIDs, but are less likely to harm the stomach lining or kidneys. May be used in combination with other medications. |
- Biologicals: etanercept (Enbrel), infliximab (Remicade)
| These injectable drugs are the first genetically engineered medications for arthritis. These anti-TNF compounds block inflammation and rapidly decrease pain and joint swelling. Enbrel is self-injected twice a week and may be used in combination with methotrexate. Remicade is administered IV at 1- to 3-month intervals. Note: Because of concerns about immune function suppression, Enbrel is recommended only for patients who are unable to tolerate methotrexate or failed to respond to at least two other DMARDs. |
- Tetracyclines:minocycline (Minocin)
| Characteristics of anti-inflammatory and immune modifier effects coupled with ability to block metalloproteinases (associated with joint destruction) have resulted in dramatic benefits in research studies. |
- - d-Penicillamine (Cuprimine)
| May control systemic effects of RA synovitis and scleroderma if other therapies have not been successful. High rate of side effects (thrombocytopenia, leukopenia, aplastic anemia) necessitates close monitoring. Note: Drug should be given between meals because drug absorption is impaired by food, as well as antacids and iron products. |
- Antacids: misoprostol (Cytotec), omeprazole (Prilosec)
| Given with NSAID agents to minimize gastric irritation and discomfort, reducing risk of GI bleed. |
- - Codeine-containing medications
| Although narcotics are generally contraindicated because of chronic nature of condition, short-term use of these products may be required during periods of acute exacerbation to control severe pain. |
Assist with physical therapies such as paraffin glove, whirlpool baths. | Provides sustained heat to reduce pain and improve ROM of affected joints. |
Apply ice or cold packs when indicated. | Cold may relieve pain and swelling during acute episodes. |
Instruct in use and monitor effect of transcutaneous electrical nerve stimulator (TENS) unit if used. | Constant low-level electrical stimulus blocks transmission of pain sensations. |
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